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TMSS Medical College Journal

Peer-reviewed  ·  English  ·  Published twice a year
ISSN 2309-3234 DOI 10.62948 BM&DC approved — 4-D-2016/1478
A Rare Case of Hereditary Angioedema with Long Term Suffering of the Patient for Misdiagnosis
Begum A1* , Akter S22 , Rahman P33
1
Dr. Aeysha Begum, Assistant Profesor, Department of Gynaccology and Obstetrics, Khawja Yunus Ali Medical College, Sirajganj
2
Dr. Sheuly Akter, Associate Professor, Deparment of Gynaccology and Obstetrics, Khawja Yunus Ali Medical College, Sirajganj
3
Dr. Parvin Rahman, Professor, Department of Gynaccology and Obsitrics, Khawja Yunus Ali Medical College, Sirajganj
Corresponding Author
Dr. Aeysha Begum
Assistant Profesor, Department of Gynaccology and Obstetrics, Khawja Yunus Ali Medical College, Sirajganj
Abstract
Angioedema is a rare disease with low prevalence (1 to 9 cases per 100,000 people). It results from deficiency or dysfunction of the serum Complement I-esterase inhibitor (C1-INH). Angioedema can be genetic or acquired. Acquired angioedema (AAE) is due to an acquired deficiency of C1-INH, caused by cither consumption (type 1) or inactivation (type 2) of CIINH. There are two types of hereditary angioedema (HAE): type | (85%), type II (15%), and both typically presents with recurrent episodes of facial ocdema, and ocdema of mucosa of the gastrointestinal tract and upper airways. The onset of ‘symptoms can occur at any age, but is most common in childhood and adolescence. Early treatment is crucial 10 prevent complications and reduce intensity of oedema. Preventive approaches include antifibrinolytics, danazol and avoidance of possible trigger. The condition frequently goes untreated for many years duc to insufficient clinical awareness. Consequently, there is a significant necessity to enhance clinical awareness and diagnostic capabilities to improve the detection and management of HAE
Keywords
Autosomal dominant Hereditary angioedema Complement C4 Serum C1 inhibitor Facial swelling.
Introduction
Hereditary angioedema (HAE) is an uncommon genetic condition resulting from the deficiency or dysfunction of the serum CI esterase inhibitor. Prevalence of HAE ranges from 1 in 10,000 to | in 150,000 and has no differences based on sex or ethnicity’. HAE typically presents with recurrent episodes of angioedema affecting the gastrointestinal tract, skin, and/orupper respiratory tract”, The diagnosis of HAE (type I or II) is usually done by clinical history and physical examination suggestive of the condition, along with significantly low levels of complement C4 in complement tests on investigation * 3, A family history of angioedema is a strong clue for the diagnosis. As HAE is rare, and symptoms often resemble other conditions, establishing a diagnosis of HAE can be difficult This case also highlights how skin-related symptoms and signs can provide important clues in diagnosing recurrent episodes of abdominal pain.
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